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109

CASO CLÍNICO

Actualidad

Médica

A C T U A L I D A D

M É D I C A

www.actualidadmedica.es

©2017.Actual.Med.Todoslosderechosreservados

A case of dilated cardiomyopathy

in a venezuelan patient with polymyositis

Abstract

Polymyositis is an autoimmune disease, characterized by the inflammation of muscular tissues. It belongs to

the family of inflammatory myopathies, along with dermatomyositis, inclusion-bodies myositis and necrotizing

autoimmune myositis. In this case report we present a 39 year old male patient admitted to the Bejuma District

Hospital in October of 1991, where the patient is evaluated for 21 days. Electromyography, skeletal muscle biopsies,

and further lab tests were performed; posing a diagnostic impression of Polymyositis. After the analysis of these

results, the patient is discharged with pharmacological treatment; however, he denies taking the medications.

In March of 2007 patient is seen at the Bejuma District Hospital after complaining of respiratory distress during

mild efforts, concomitantly non-productive cough and palpitations. It is concluded that the rate of cardiovascular

deterioration without constant medical treatment is secondary to untreated Polymyositis.

Resumen

La polimiositis es una enfermedad autoinmune, caracterizada por la inflamación de los tejidos musculares.

Pertenece a la familia de las miopatías inflamatorias, junto con dermatomiositis, miositis de cuerpos de inclu-

sión y miositis autoinmune necrotizante. En este caso presentamos un paciente varón de 39 años admitido

en el Hospital del Distrito de Bejuma en octubre de 1991, donde el paciente es evaluado durante 21 días. Se

realizaron electromiografía, biopsias del músculo esquelético y más pruebas de laboratorio; lo que supone

una impresión diagnóstica de Polimiositis. Después del análisis de estos resultados, el paciente es dado de alta

con tratamiento farmacológico; sin embargo, niega tomar los medicamentos. En marzo de 2007 el paciente es

visto en el Hospital del Distrito de Bejuma después de quejarse de dificultad respiratoria durante los esfuerzos

suaves, concomitantemente tos no productiva y palpitaciones. Se concluye que la tasa de deterioro cardiovas-

cular sin tratamiento médico constante es secundaria a la polimiositis no tratada.

Miguel Bayones MD

1

, Ana Ramírez MD

1

, Jose Ruiz MD

2

, Carlos Sierra MS

3

, Claudio

Tombazzi

4

1

Internal Medicine Service, Bejuma District Hospital, Bejuma, Venezuela

2

Research Service, Memphis VA Medical Center, Memphis, USA

3

University of Kentucky School of Medicine, University of Kentucky, Lexington, USA

4

University of Tennessee School of Medicine, University of Tennessee, Memphis, USA

Enviado: 15-04-2017

Revisado: 20-05-2017

Aceptado: 10-06-2017

Jose Ruiz, MD

Av. 90. Urb. Las Chimeneas

Edif. San Andres VII, Apt. 10-C. Valencia, Edo Carabobo, Venezuela.

Telephone: +582418420996

E-mail Address:

josei_16@hotmail.com

Palabras clave: Polimiositis,

miocardiopatía dilatada,

miopatía inflamatoria

Keywords: Polymyositis, dilated

cardiomyopathy, inflammatory

myopathy

Un caso de cardiomiopatía dilatada en un paciente venezolano

con polimiositis

DOI: 10.15568/am.2017.801.cc05

Actual. Med.

2017; 102: (801): 109-111

INTRODUCTION

Polymyositis is an autoimmune disease which belongs to the

family of inflammatory myopathies, along with dermatomyositis,

inclusion-bodiesmyositis and necrotizing autoimmunemyositis (1,2).

It affects the muscle tissue through an inflammatory and necrotizing

process that results in progressive weakness. It may also affect the

cardiac muscle. The average age of presentation ranges between 45

and 60 years, and it seldom occurs during childhood (3).

Due to its uncommon nature; we present the study of a clinical

case in which a patient with a diagnosis of Polymyositis attends to

the Bejuma District Hospital, State of Carabobo, Venezuela, with

manifestations of dilated cardiomyopathy class III.

CLINICAL CASE

Case of a 39-year-old patient from Carabobo, Venezuela,

that started exhibiting symptoms of the disease in August of

1991. The patient begins to exhibit proximal muscle weakness

in the lower extremities in a progressive and symmetric

manner. This progressive muscle weakness limits the patient’s